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Norm’s note: The first that I heard about a possible association between prion disease (i.e., spongiform encephalopathy) and the ‘spike protein’ was in two interviews that recently surfaced, one with Dr. Richard Fleming and the other with Pr Luc Montagnier.  

Now I have a reference, thanks to the Hart group, to an independent peer reviewed study by J. Bart Classen, MD, that corroborates the claims of Fleming and Montagnier, in that prion disease is a definite long term danger associated with the Pfizer COVID-19 vaccine.  

If you are thinking of encouraging or having your children vaccinated with ANY of the vaccines, you would probably do well to reconsider in light of this information.

Source: COVID-19 RNA Based Vaccines and the Risk of Prion Disease 

The abstract of the research article reads as follows : 


Development of new vaccine technology has been plagued with problems in the past. The current RNA based SARS-CoV-2 vaccines were approved in the US using an emergency order without extensive long term safety testing. In this paper the Pfizer COVID-19 vaccine was evaluated for the potential to induce prion-based disease in vaccine recipients. The RNA sequence of the vaccine as well as the spike protein target interaction were analyzed for the potential to convert intracellular RNA binding proteins TAR DNA binding protein (TDP-43) and Fused in Sarcoma (FUS) into their pathologic prion conformations. The results indicate that the vaccine RNA has specific sequences that may induce TDP-43 and FUS to fold into their pathologic prion confirmations. In the current analysis a total of sixteen UG tandem repeats (ΨGΨG) were identified and additional UG (ΨG) rich sequences were identified. Two GGΨA sequences were found. Potential G Quadruplex sequences are possibly present but a more sophisticated computer program is needed to verify these. Furthermore, the spike protein, created by the translation of the vaccine RNA, binds angiotensin converting enzyme 2 (ACE2), a zinc containing enzyme. This interaction has the potential to increase intracellular zinc. Zinc ions have been shown to cause the transformation of TDP-43 to its pathologic prion configuration. The folding of TDP-43 and FUS into their pathologic prion confirmations is known to cause ALS, front temporal lobar degeneration, Alzheimer’s disease and other neurological degenerative diseases. The enclosed finding as well as additional potential risks leads the author to believe that regulatory approval of the RNA based vaccines for SARS-CoV-2 was premature and that the vaccine may cause much more harm than benefit. (Norm’s emphasis)